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Further, energy mobilization and expenditure were equivalent, with genotypes exhibiting identical circadian activity cycles (Figure 4E) and thermoregulatory reflexes to a cold (4°C) environment (ref. Thus, differences in these other adipogenic systems were not observed between the genotypes, establishing increased calorie consumption as the cause of the excess adiposity observed in Gucy2c (A) Free fatty acid content of feces of 3- to 4-month-old mice raised on HCD (n = 16).Scattergram points represent data for individual mice.

This endocrine prohormone signal is selectively decoded centrally by specific proteolytic liberation of uroguanylin, but not guanylin, unexpectedly activating neuronal GUCY2C and downstream anorexigenic pathways.Department of Pharmacology and Experimental Therapeutics, Division of Biostatistics, Thomas Jefferson University, Philadelphia, Pennsylvania, USA. in: JCI | Pub Med | Google Scholar Department of Pharmacology and Experimental Therapeutics, Division of Biostatistics, Thomas Jefferson University, Philadelphia, Pennsylvania, USA. in: JCI | Pub Med | Google Scholar Department of Pharmacology and Experimental Therapeutics, Division of Biostatistics, Thomas Jefferson University, Philadelphia, Pennsylvania, USA. in: JCI | Pub Med | Google Scholar Department of Pharmacology and Experimental Therapeutics, Division of Biostatistics, Thomas Jefferson University, Philadelphia, Pennsylvania, USA. in: JCI | Pub Med | Google Scholar Department of Pharmacology and Experimental Therapeutics, Division of Biostatistics, Thomas Jefferson University, Philadelphia, Pennsylvania, USA. in: JCI | Pub Med | Google Scholar Department of Pharmacology and Experimental Therapeutics, Division of Biostatistics, Thomas Jefferson University, Philadelphia, Pennsylvania, USA. in: JCI | Pub Med | Google Scholar Department of Pharmacology and Experimental Therapeutics, Division of Biostatistics, Thomas Jefferson University, Philadelphia, Pennsylvania, USA. in: JCI | Pub Med | Google Scholar Department of Pharmacology and Experimental Therapeutics, Division of Biostatistics, Thomas Jefferson University, Philadelphia, Pennsylvania, USA. in: JCI | Pub Med | Google Scholar Department of Pharmacology and Experimental Therapeutics, Division of Biostatistics, Thomas Jefferson University, Philadelphia, Pennsylvania, USA. in: JCI | Pub Med | Google Scholar Department of Pharmacology and Experimental Therapeutics, Division of Biostatistics, Thomas Jefferson University, Philadelphia, Pennsylvania, USA. in: JCI | Pub Med | Google Scholar Prouroguanylin is a gastrointestinal paracrine signal and prohormone that is secreted after nutrient ingestion. show that prouroguanylin is converted to uroguanylin in the CNS, which can activate guanylyl cyclase 2C (GUCY2C) receptors in the brain to reduce food intake in mice.



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